Growth arrest–specific protein 6 protects against renal ischemia–reperfusion injury
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منابع مشابه
Correction: Prion Protein Protects against Renal Ischemia/Reperfusion Injury
There are errors in the author affiliations for Fan Zhang. The affiliations should appear as shown here: Fan Zhang 13, 15 2 Department of Pathology, Case Western Reserve University/University Hospitals Case Medical Center, Cleveland, Ohio, United States of America, 13 Department of Neurosurgery, Shandong University, Shandong provincial hospital, Jinan, The People’s Republic of China, 15 Departm...
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The cellular prion protein (PrPC), a protein most noted for its link to prion diseases, has been found to play a protective role in ischemic brain injury. To investigate the role of PrPC in the kidney, an organ highly prone to ischemia/reperfusion (IR) injury, we examined wild-type (WT) and PrPC knockout (KO) mice that were subjected to 30-min of renal ischemia followed by 1, 2, or 3 days of re...
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Relaxin, a pregnancy hormone, has antiapoptotic and anti-inflammatory properties. The aim of this study was to determine the effects of relaxin on ischemia-reperfusion (IR)-induced acute kidney injury. Male rats underwent unilateral nephrectomy and contralateral renal IR (45 min of renal pedicle clamping). Rats were divided into three groups: 1) sham group, 2) IR group, and 3) IR-RLX group (rat...
متن کاملHuman liver-type fatty acid-binding protein protects against tubulointerstitial injury in aldosterone-induced renal injury.
To demonstrate the renoprotective function of human liver-type fatty acid-binding protein (hL-FABP) expressed in proximal tubules in aldosterone (Aldo)-induced renal injury, hL-FABP chromosomal transgenic (Tg) and wild-type (WT) mice received systemic Aldo infusions (Tg-Aldo and WT-Aldo, respectively) were given 1% NaCl water for 28 days. In this model, elevation of systolic blood pressure, mon...
متن کاملN-acetylcysteine protects against renal injury following bilateral ureteral obstruction
BACKGROUND Obstructive nephropathy decreases renal blood flow (RBF) and glomerular filtration rate (GFR), causing tubular abnormalities, such as urinary concentrating defect, as well as increasing oxidative stress. This study aimed to evaluate the effects of N-acetylcysteine (NAC) on renal function, as well as on the protein expression of aquaporin 2 (AQP2) and endothelial nitric oxide synthase...
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ژورنال
عنوان ژورنال: Journal of Surgical Research
سال: 2015
ISSN: 0022-4804
DOI: 10.1016/j.jss.2015.05.049